Release date: 2015-10-27
Recently, researchers from Australia pointed out that a simple blood test can predict whether a healthy person may die of pneumonia or sepsis in the next 14 years. According to an analysis of 10,000 people, the researchers discovered a molecular byproduct of inflammation called GlycA, which may predict premature death from infection.
The disease can be detected by a simple blood test, which is well known to us. For example, scientists have shown that simple blood tests can help diagnose early Alzheimer's disease and can also be used as an early cancer diagnosis tool.
Recently, researchers from Australia pointed out that a simple blood test can predict whether a healthy person may die of pneumonia or sepsis in the next 14 years. According to an analysis of 10,000 people, the researchers discovered a molecular byproduct of inflammation called GlycA, which may predict premature death from infection.
The results of the study, published in the Cell issue of Cell, October 22, show that high levels of GlycA in the blood can indicate a chronic inflammatory condition that may be due to low levels of chronic infection, or too Active immune response. This inflammation can damage the body and may make individuals more susceptible to serious infections.
Michael Inouye, a senior author of the paper, and the University of Melbourne pointed out: "As biomedical researchers, we want to help people. I can think of some more important things, that is, to identify those seemingly healthy, actually may People at increased risk of disease and death. We want to reduce these risks, and we need to understand what blood biomarkers of the disease's risk actually tell us."
Inouye and his colleagues point out that more research is needed to clarify "the mechanism by which GlycA is linked to inflammation and premature death, and whether clinically detecting GlycA levels is likely to be approved."
Johannes Kettunen, co-author of this article, University of Oulu, Finland, and Finnish National Institute for Health and Welfare, said: “We still have a lot of work to see if we can reduce the risk in some way.†For example, in order to plan a course of treatment For treatment, researchers need to know that high levels of GlycA are a result of chronic, low-level microbial infections or an abnormal response to their own inflammatory response.
These findings may lay the groundwork for many other studies that explore the role of GlycA in vivo. Inouye said: “The more health records we have and the high-quality genomic data we have long-term follow-up, the better models and predictions we can make. This study is a collaborative study of volunteers, doctors, technicians, and data scientists. Examples, but we have to do more work, and large-scale strategic interdisciplinary initiatives are necessary."
Source: Biopass
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