I. Symptoms In the early stages of disease onset, many round or near-circular acupuncture-like, purplish-red patches appeared on the leaves, and after 2 days, the patches were depressed. After 5 days, the lesions expanded and appeared black. The center often has a bright spot that is a spore plate of germs. When the lesions become contiguous, they become polygonal spots or round spots. When they are severe, all the leaves become dark, causing the seedlings to die. The dead seedlings of early deciduous trees are short, distorted, and their stems and leaves turn black. The lesions on the petioles and tender stems were fusiform, dark brown, and slightly sunken in the center.
Second, the incidence of law Poplar rot disease is caused by the fungus. The pathogens overwinter in diseased leaves or diseased shoots. In the spring of the following year, conidia were produced when the conditions were appropriate and became the primary source of infestation. Onset began in May, and onset in July-August and stopped in September. Therefore, the high-temperature and high-humidity rainy season is more severe, and the heavily populated land, low-lying land, weak seedlings, or seedlings are too dense and the seedbed is too wet to be susceptible to the disease.
Third, prevention and control methods 1 combined with the actual introduction of production, breeding and cultivation of disease-resistant, fast-growing fine tree species. Use a variety of species layout planting to avoid large areas of pure forest. 2 Select well-drained nursery plots, avoid continuous cropping, change crops for seedlings in a planned manner, and use 75% pentachloronitrobenzene or 25% doxorubicin plus dixon for soil disinfection. 3 Properly dense planting to improve the ventilation and light conditions of the seedlings. 4 According to the local disease occurrence, 65% of zeocin zinc 125-250 times can be sprayed in the early stage of the disease, and sprayed 2-3 times.
Second, the incidence of law Poplar rot disease is caused by the fungus. The pathogens overwinter in diseased leaves or diseased shoots. In the spring of the following year, conidia were produced when the conditions were appropriate and became the primary source of infestation. Onset began in May, and onset in July-August and stopped in September. Therefore, the high-temperature and high-humidity rainy season is more severe, and the heavily populated land, low-lying land, weak seedlings, or seedlings are too dense and the seedbed is too wet to be susceptible to the disease.
Third, prevention and control methods 1 combined with the actual introduction of production, breeding and cultivation of disease-resistant, fast-growing fine tree species. Use a variety of species layout planting to avoid large areas of pure forest. 2 Select well-drained nursery plots, avoid continuous cropping, change crops for seedlings in a planned manner, and use 75% pentachloronitrobenzene or 25% doxorubicin plus dixon for soil disinfection. 3 Properly dense planting to improve the ventilation and light conditions of the seedlings. 4 According to the local disease occurrence, 65% of zeocin zinc 125-250 times can be sprayed in the early stage of the disease, and sprayed 2-3 times.
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