Knocking out an aliphatic cell or helping to heal bone damage

:2019-06-05

Patients with multiple myeloma may develop bone damage that cannot heal even when the condition is relieved. To understand the mechanisms behind these osteolytic lesions, researchers such as Liu Huan of the University of Texas MD Anderson Cancer Center studied samples from patients in remission.

To demonstrate that myeloma cells affect adipocyte-mediated bone resorption, they performed xenograft experiments in mouse and in vitro co-cultures.

This mechanism involves EZH2-dependent inhibition of peroxisome proliferator-activated receptor gamma (PPAR-γ), which results in knockdown of mouse adipocyte EZH2 to reduce bone lesions after multiple myeloma remission. This discovery reveals a new way to heal bone damage.

Source: Technology Daily

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